Conolidine’s analgesic outcomes stem from its conversation with non-opioid pain pathways. Unlike opioids, which bind to µ-opioid receptors within the central nervous method, conolidine modulates alternate molecular targets. A Science Advancements research observed that conolidine interacts Along with the atypical chemokine receptor ACKR3/CXCR7,

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Importantly, these receptors ended up observed to are actually activated by a variety of endogenous opioids in a focus comparable to that noticed for activation and signaling of classical opiate receptors. Consequently, these receptors have been located to possess scavenging exercise, binding to and reducing endogenous amounts of opiates available

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